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However, this method of entry means it can bypass the liver (unlike c-17 alpha alkylated steroids), thus entering the bloodstream immediatelyafter initiation of therapy. Therefore, it is necessary to provide an injection of the steroid to begin therapy. In contrast, testosterone administration to non-fasting patients (and to subjects with the body mass index (BMI) higher than 30 kg/m(2) > 40 kg/m(2) ) results in lower absolute levels of free testosterone and is more likely to lead to an increase in body weight compared with non-fasting subjects. When combined with insulin, it results in a decrease in body weight compared with a placebo. Thus, a combined testosterone/insulin injection is the recommended method of treatment of diabetes. Since obesity is a risk factor for the development of type 2 diabetes mellitus in men and women, the aim of treatments for this condition is to change obesity status to normal levels by decreasing the incidence of obesity (and weight gain) in the general population. Diabetes mellitus is a metabolic disorder occurring mainly in the elderly who have abnormal glucose metabolism due to excessive accumulation of fat in the pancreas, abdominal visceral fat, and the liver (see ). A large proportion of non-diabetic people who are overweight or obese die from these conditions. According to the World Health Organization (WHO), the prevalence of diabetes at age 65 is 1.3% while that of type 2 diabetes is 1%. This percentage increases to 2.6% in middle-aged individuals, 3.8% in obese subjects and 5.3% in obese non-diabetic subjects. The cause of diabetic complications is a complex and multifactorial process. The mechanisms that underlie the diabetes process include hormonal changes that can lead to an overgrowth or underproduction of insulin, and an abnormal release of cortisol, a key hormone responsible for the release of insulin from the liver. The pathogenesis of non-insulin dependent diabetes mellitus (NIDDM), also known as idiopathic diabetes mellitus (IDM), can be divided into four stages that may be considered as sequential, with a fourth stage (non-Mendelian) which seems to be rare. The development of non-Mendelian-type diabetes (NMDDM) usually begins in adult life. The development is initiated primarily during adolescence by a gradual increase of plasma free fatty acids until the mid-thirties when high total daily energy expenditure is reached. A series of physiological changes, including an increased hepatic glucose production and insulin resistance, follow and eventually result in a dramatic increase in Related Article: